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::::I've tried to speak with her, and even discussed the matter of Rahab and Boaz with her, but she dismisses what I say because I am younger and simply says that I am not reading my Bible closely enough. Perhaps printing off the article would be a good idea. [[User:DanH|DanH]] 02:43, 28 April 2008 (EDT)
::::I've tried to speak with her, and even discussed the matter of Rahab and Boaz with her, but she dismisses what I say because I am younger and simply says that I am not reading my Bible closely enough. Perhaps printing off the article would be a good idea. [[User:DanH|DanH]] 02:43, 28 April 2008 (EDT)
:::::A very interesting paper Phillip but it is mistaken about genetics of melanin. I am unsure if this was to simplify the paper or not but it is much more complicated than the paper lets on. For instance there are two types of melanin, phelomelanin and eumelanin. One gives a reddish hue while the other gives a dark brown hue. This is important since the amount of melanin is encoded on six genes, not two as the paper presents; it is also controlled by several alleles allowing for great variance to the possible hues. This is all greatly complicated by the fact that the genes for melanin are neither dominant nor recessive allowing for incomplete dominance, thus allowing for shades within a color. The comment about the epicanthic fold is really a stretch, it is genetic in nature and not caused by more fat. There is an actual physiological difference in the eye lid to cause the fold. I must say the article is correct in its assumption of how melanogenesis affects the coloring of skin. We all have a basal level of melanin and an activated level. The basal level is what determines race while the active level is based on UV exposure. Both the active and basal levels are dictated genetically. Just my two cents.--[[User:Able806|Able806]] 09:22, 28 April 2008 (EDT)
:::::A very interesting paper Phillip but it is mistaken about genetics of melanin. I am unsure if this was to simplify the paper or not but it is much more complicated than the paper lets on. For instance there are two types of melanin, phelomelanin and eumelanin. One gives a reddish hue while the other gives a dark brown hue. This is important since the amount of melanin is encoded on six genes, not two as the paper presents; it is also controlled by several alleles allowing for great variance to the possible hues. This is all greatly complicated by the fact that the genes for melanin are neither dominant nor recessive allowing for incomplete dominance, thus allowing for shades within a color. The comment about the epicanthic fold is really a stretch, it is genetic in nature and not caused by more fat. There is an actual physiological difference in the eye lid to cause the fold. I must say the article is correct in its assumption of how melanogenesis affects the coloring of skin. We all have a basal level of melanin and an activated level. The basal level is what determines race while the active level is based on UV exposure. Both the active and basal levels are dictated genetically. Just my two cents.--[[User:Able806|Able806]] 09:22, 28 April 2008 (EDT)
:::: From the link that you apparently read only superficially:
{{QuoteBox|We know that skin colour is governed by more than one pair of genes. For simplicity, let’s assume there are only two,<sup>9</sup>...<br />
At position A we could have a pair such as M<sub>A</sub>M<sub>A</sub>,, M<sub>A</sub>m<sub>A</sub> or m<sub>A</sub>m<sub>A</sub><sup>11</sup>...<br />
If three gene pairs were involved, seven levels of melanin would be possible. ...<br />
We need to remember, of course, that the way in which genes express themselves is much more complex than this simplified picture. For example, sometimes certain genes are linked together. However, the basic point is unaffected.<br />
<small><small>9. This simplification is not done to help our case—the more genes there are, the easier it is
to have a huge range of ‘different’ colours. The principle involved can be understood by
using two as an example.<br />
11. For the technically minded, this type of genetic expression, where allele dosage affects the
trait, is called partial dominance.</small></small>}}
:::: [[User:Philip J. Rayment|Philip J. Rayment]] 10:27, 28 April 2008 (EDT)