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==Pathophysiology==
 
==Pathophysiology==
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Acinetobacter species are non-motile [[bacteria]] which appear as gram-negative coccobaccili in pairs on microscopic study.  There are 25 different strains<ref>Collaborative effort of the DHSD, AFIOH, DHCC, NEHC, GEIS and USACHPPM. [http://www.pdhealth.mil/downloads/Acinetobacter_FS_120804.pdf ''Acinetobacter Infections -- Information for Servicemembers and Their Families'']</ref>, though ''A. Baumanni'' is responsible for 80% of infections.  All species are classified as non-lactose fermenting.
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Acinetobacter species are non-motile [[bacteria]] which appear as gram-negative coccobaccili in pairs on microscopic study.  There are 25 different strains,<ref>Collaborative effort of the DHSD, AFIOH, DHCC, NEHC, GEIS and USACHPPM. [http://www.pdhealth.mil/downloads/Acinetobacter_FS_120804.pdf ''Acinetobacter Infections -- Information for Servicemembers and Their Families'']</ref> though ''A. Baumanni'' is responsible for 80% of infections.  All species are classified as non-lactose fermenting.
    
==Clinical significance==
 
==Clinical significance==
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''Acinetobacter'' causes a variety of [[disease|diseases]], ranging from nosocomial (hospital-aquired) [[pneumonia]] to serious wound infections.  Symptoms of a wound infection include redness and/or heat at the site, fever, chills, purulent or odd-colored drainage, and pain.  Although not prevolent in the general population, ''Acinetobacter'' can cause severe, life-threatening illness in immunocompromised patients.<ref>Ulrike Gerisher (2008). [http://www.horizonpress.com/acineto ''Acinetobacter Molecular Biology'']</ref>
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''Acinetobacter'' cause a variety of [[disease]]s, ranging from nosocomial (hospital-acquired) [[pneumonia]] to serious wound infections.  Symptoms of a wound infection include redness and/or heat at the site, fever, chills, purulent or odd-colored drainage, and pain.  Although not prevalent in the general population, ''Acinetobacter'' can cause severe, life-threatening illness in immunocompromised patients.<ref>Ulrike Gerisher (2008). [http://www.horizonpress.com/acineto ''Acinetobacter Molecular Biology'']</ref>
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==Treatment==
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Acinetobacter is resistant to many different strains of antibiotics&mdash;penicillins, cephalosporins, flourquinolones, and aminoglycosides.  Luckily, due to its typically non-pathogenic nature, its resistance does not have much clinical significance.  However, in the case of an infection, the first line of defense are the carbapenems&mdash; imipenem, meropenem, etc.  Recently, there has been a worrying increase in carbapenem resistance, leading to very little lines of defense for Acinetobacter.
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==Iraq War==
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In November 2004, according to the [[CDC]], it was found that many troops in military hospitals had blood infections of multi-drug resistance to Acinetobacter.  This was found in both [[Afghanistan]] and [[Iraq]].  This shows the need for more funding and support to our medical centers overseas.
    
==References==
 
==References==
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