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Talk:Flaws in Richard Lenski Study (view source)
Revision as of 16:46, July 15, 2008
, 16:46, July 15, 2008→Expertise in Statistics
: OK, here's point 4 in layman's terms: Lenski's hypotheses of a mutation rate imply that a ten-fold increase in sample size should result in a ten-fold increase in mutations. But it doesn't. In fact, a nearly ten-fold increase in sample size results in only a slight increase in mutations in Lenski's data. These data, as presented by Lenski in his paper, suggest (if properly interpreted) that there is no mutation rate at all. Rather, these data are more consistent with occasional contamination, broadly defined.--[[User:Aschlafly|Aschlafly]] 23:15, 14 July 2008 (EDT)
: OK, here's point 4 in layman's terms: Lenski's hypotheses of a mutation rate imply that a ten-fold increase in sample size should result in a ten-fold increase in mutations. But it doesn't. In fact, a nearly ten-fold increase in sample size results in only a slight increase in mutations in Lenski's data. These data, as presented by Lenski in his paper, suggest (if properly interpreted) that there is no mutation rate at all. Rather, these data are more consistent with occasional contamination, broadly defined.--[[User:Aschlafly|Aschlafly]] 23:15, 14 July 2008 (EDT)
::Except that the growth pattern of the Cit+ cells in the third replay experiment demonstrated that there were no Cit+ cells in the cultures at the time of plating. Control plates with a mix of Cit- and Cit+ cells were done which showed rapid development of Cit+ colonies, whereas the experimental plates did not show any Cit+ colonies for at least 8 days and up to 28 days. If you believe that the plates became randomly contaminated during the course of the incubation, rather than at the initial plating, the post-plating contamination should have affected all generations equally, but it did not--[[User:Brossa|Brossa]] 12:46, 15 July 2008 (EDT)