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In June, 2010, a team including Shi Zhengli, published a paper.  It described research to understand the susceptibility of angiotensin converting enzyme 2 ACE-2 proteins, of different bat species to the S Protein of the SARS virus.  In the experiments, they also modified key amino acid codons to mutate the bats ACE-2 to examine compatibility with the SARS S Protein.  This paper demonstrated their awareness of the special relationship between the S Protein and the ACE-2 receptor.  It also signified that Shi had unearthed the passageway for coronaviruses into human bodies.
 
In June, 2010, a team including Shi Zhengli, published a paper.  It described research to understand the susceptibility of angiotensin converting enzyme 2 ACE-2 proteins, of different bat species to the S Protein of the SARS virus.  In the experiments, they also modified key amino acid codons to mutate the bats ACE-2 to examine compatibility with the SARS S Protein.  This paper demonstrated their awareness of the special relationship between the S Protein and the ACE-2 receptor.  It also signified that Shi had unearthed the passageway for coronaviruses into human bodies.
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[[File:Daszak shi 0 0.jpg|left|30px|thumb|[[Peter Daszak]] (right) toasting Shi Zhengli (left).]]
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[[File:Daszak shi 0 0.jpg|left|300px|thumb|[[Peter Daszak]] (right) toasting Shi Zhengli (left).]]
 
In October, 2013, Shi and her team published a paper in ''Nature''.  They claimed a breakthrough in coronavirus research.  They successfully isolated three viruses from bats, one of which had a S Protein that integrated with human ACE-2 receptors.  This effectively demonstrated the direct human infection of SARS-like viruses to humans, without the need of an intermediate host.
 
In October, 2013, Shi and her team published a paper in ''Nature''.  They claimed a breakthrough in coronavirus research.  They successfully isolated three viruses from bats, one of which had a S Protein that integrated with human ACE-2 receptors.  This effectively demonstrated the direct human infection of SARS-like viruses to humans, without the need of an intermediate host.
  
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